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Spedding, M. and Sebban, C. and Jay, T.M. and Rocher, C. and Tesolin-Decros, B. and Chazot, P. and Schenker, E. and Sz{\'e, Phenotypical Screening on Neuronal Plasticity in Hippocampal-Prefrontal Cortex Connectivity Reveals an Antipsychotic with a Novel Profile, Cells, 11, (7), 2022,
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Zaaimi B, Turnbull M, Hazra A, Wang Y, Gandara C, McLeod F, McDermott EE, Escobedo-Cousin E, Idil AS, Bailey RG, Tardio S, Patel A, Ponon N, Gausden J, Walsh D, Hutchings F, Kaiser M, Cunningham MO, Clowry GJ, LeBeau FEN, Constandinou TG, Baker SN, Donaldson N, Degenaar P, O'Neill A, Trevelyan AJ, Jackson A., Closed-loop optogenetic control of the dynamics of neural activity in non-human primates., Nature biomedical engineering, 2022,
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Cunningham MO., Cross Talk proposal: Human-derived brain tissue is a better epilepsy model than animal-based approaches., The Journal of physiology, 600, (21), 2022, p4569-4574 ,
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Lopez-Rodriguez, A.B. and Hennessy, E. and Murray, C.L. and Nazmi, A. and Delaney, H.J. and Healy, D. and Fagan, S.G. and Rooney, M. and Stewart, E. and Lewis, A. and de Barra, N. and Scarry, P. and Riggs-Miller, L. and Boche, D. and Cunningham, M.O. and Cunningham, C., Acute systemic inflammation exacerbates neuroinflammation in Alzheimer's disease: IL-1β drives amplified responses in primed astrocytes and neuronal network dysfunction, Alzheimer's and Dementia, 17, (10), 2021, p1735-1755 ,
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Morris G and Rowell R and Cunningham M, Limitations of animal epilepsy research models: Can epileptic human tissue provide translational benefit?, ALTEX, 2021,
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Chan F, Lax NZ, Voss CM, Aldana BI, Whyte S, Jenkins A, Nicholson C, Nichols S, Tilley E, Powell Z, Waagepetersen HS, Davies CH, Turnbull DM, Cunningham MO, The role of astrocytes in seizure generation: insights from a novel in vitro seizure model based on mitochondrial dysfunction, Brain, 142, (2), 2019, p391 - 411,
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Sánchez RG, Parrish RR, Rich M, Webb WM, Lockhart RM, Nakao K, Ianov L, Buckingham SC, Broadwater DR, Jenkins A, de Lanerolle NC, Cunningham M, Eid T, Riley K, Lubin FD., Human and rodent temporal lobe epilepsy is characterized by changes in O-GlcNAc homeostasis that can be reversed to dampen epileptiform activity., Neurobiology of disease, 124, 2019, p531-543 ,
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